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However, GHI IS contagious, typically through skin-to-skin contact with an infected area.

The method of transmission is may occur through an active herpes blister on one person with a broken area of skin on the other person.

Obviously such matters would be very difficult to verify.

The herpes virus does not survive outside the body for more than about 10 seconds, and although it can survive for slightly longer in warm, damp conditions, it dies very quickly once exposed to the air.

It is not the “skin” that is infected but rather the nerve cell.

When the infection becomes active again in the nucleus of the nerve cell, the viral particles flow back out, down the nerve, and out through little blisters that form in the skin.

I will refrain from calling GHI the more typical name “HSV-2” for the reason discussed above.

Herpes is a DNA-type virus, inserting its DNA directly into the dendritic nerve endings of the skin, which then leads along nerve fibers to the nucleus of the nerve cell.

Once the viral information is inserted into the cell’s nucleus, this blending of viral genetic information with human genetic information is permanent.

Probably Human Papillomavirus (HPV) infections outnumber GHI (see the article on HPV) in both growth rate and probably in absolute numbers.

Generally GHI is not considered to be extremely contagious.

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